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Attention deficit hyperactivity disorder (ADHD) is a highly heritable neuropsychiatric condition, but due to its complexity it has been challenging to identify genes underlying this disorder. Our genetics group at K.G.Jebsen center performed the first genome-wide association analyses of ADHD in an ethnically homogeneous Norwegian population.
Taken together with previous findings, our results point to a spectrum of biological mechanisms underlying the symptoms of ADHD, such as gene expression and cell adhesion, pointing towards neurogenesis and inflammation. This study confirms existing targets and provides new ones for further genetic exploration as well as treatment and prevention of this complex disorder.
Text by Tetyana Zayats (researcher, KGJN)